Induction of interferon-λ contributes to TLR3 and RIG-I activation-mediated inhibition of herpes simplex virus type 2 replication in human cervical epithelial cells-Reference-Cited by-同舟云学术

Induction of interferon-λ contributes to TLR3 and RIG-I activation-mediated inhibition of herpes simplex virus type 2 replication in human cervical epithelial cells

Published:2015-10-26 Issue:12 Volume:21 Page:917-929
ISSN:1360-9947
Container-title:Molecular Human Reproduction
language:en
Short-container-title:Mol. Hum. Reprod.

Author:

Zhou Li,Li Jie-Liang,Zhou Yu,Liu Jin-Biao,Zhuang Ke,Gao Jian-Feng,Liu Shi,Sang Ming,Wu Jian-Guo,Ho Wen-Zhe

Funder

National Natural Science Foundation of China

Fundamental Research Funds for the Central Universities

China Postdoctoral Science Foundation

Development Program of China

National Institute on Drug Abuse (NIDA)

Hubei Key Laboratory of Wudang Local Chinese Medicine Research

Publisher

Oxford University Press (OUP)

Subject

Cell Biology,Developmental Biology,Obstetrics and Gynecology,Genetics,Molecular Biology,Embryology,Reproductive Medicine

Link

http://academic.oup.com/molehr/article-pdf/21/12/917/3308478/gav058.pdf

Reference56 articles.

1. Recognition of double-stranded RNA and activation of NF-κB by Toll-like receptor 3

2. Type III IFNs: New layers of complexity in innate antiviral immunity

3. Lambda Interferon (IFN-λ), a Type III IFN, Is Induced by Viruses and IFNs and Displays Potent Antiviral Activity against Select Virus Infections In Vivo

4. Toll‐Like Receptor (TLR)–3, but Not TLR4, Agonist Protects against Genital Herpes Infection in the Absence of Inflammation Seen with CpG DNA

5. CpG DNA stimulates autoreactive immature B cells in the bone marrow

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