Selective activation of PI3Kα/Akt/GSK-3β signalling and cardiac compensatory hypertrophy during recovery from heart failure-Reference-Cited by-同舟云学术

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Selective activation of PI3Kα/Akt/GSK-3β signalling and cardiac compensatory hypertrophy during recovery from heart failure

Published:2009-07-24 Issue:8 Volume:11 Page:739-748
ISSN:1388-9842
Container-title:European Journal of Heart Failure
language:en
Short-container-title:European Journal of Heart Failure

Author:

Braz Julian C.¹,Gill Robert M.¹,Corbly Angela K.¹,Jones Bonita D.¹,Jin Najia¹,Vlahos Chris J.¹,Wu Qingyu²,Shen Weiqun³

Affiliation:

1. Lilly Research Laboratories; Eli Lilly and Company; Indianapolis IN 46285 USA

2. Molecular Cardiology, Nephrology and Hypertension; Cleveland Clinic; Cleveland OH 44195 USA

3. Institute of Molecular and Experimental Therapeutics; East China Normal University; Shanghai China

Publisher

Wiley

Subject

Cardiology and Cardiovascular Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1093/eurjhf/hfp094/fullpdf

Reference37 articles.

1. Signalling through the lipid products of phosphoinositide-3-OH kinase;Toker;Nature,1997

2. The phosphoinositide 3-kinase pathway;Cantley;Science,2002

3. The role of phosphoinositide-3 kinase and PTEN in cardiovascular physiology and disease;Oudit;J Mol Cell Cardiol,2004

4. The conserved phosphoinositide 3-kinase pathway determines heart size in mice;Shioi;EMBO J,2000

5. Phosphoinositide 3-kinase(p110alpha) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy;McMullen;Proc Natl Acad Sci USA,2003

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