Mitochondrial complex I defect and increased fatty acid oxidation enhance protein lysine acetylation in the diabetic heart-Reference-Cited by-同舟云学术

Mitochondrial complex I defect and increased fatty acid oxidation enhance protein lysine acetylation in the diabetic heart

Published:2015-06-22 Issue:4 Volume:107 Page:453-465
ISSN:0008-6363
Container-title:Cardiovascular Research
language:en
Short-container-title:Cardiovasc Res

Author:

Vazquez Edwin J.,Berthiaume Jessica M.,Kamath Vasudeva,Achike Olisaemeka,Buchanan Elizabeth,Montano Monica M.,Chandler Margaret P.,Miyagi Masaru,Rosca Mariana G.

Funder

Juvenile Diabetes Research Foundation International

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/107/4/453/17195062/cvv183.pdf

Reference50 articles.

1. New type of cardiomyopathy associated with diabetic glomerulosclerosis

2. Rodent models of diabetic cardiomyopathy

3. Impaired Cardiac Efficiency and Increased Fatty Acid Oxidation in Insulin-Resistant ob/ob Mouse Hearts

4. Reduced Mitochondrial Oxidative Capacity and Increased Mitochondrial Uncoupling Impair Myocardial Energetics in Obesity

5. Effect of Obesity and Insulin Resistance on Myocardial Substrate Metabolism and Efficiency in Young Women

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