5-Aza-2′-deoxycytidine causes replication lesions that require Fanconi anemia-dependent homologous recombination for repair-Reference-Cited by-同舟云学术

5-Aza-2′-deoxycytidine causes replication lesions that require Fanconi anemia-dependent homologous recombination for repair

Published:2013-04-22 Issue:11 Volume:41 Page:5827-5836
ISSN:1362-4962
Container-title:Nucleic Acids Research
language:en
Short-container-title:

Author:

Orta Manuel Luís,Calderón-Montaño José Manuel,Domínguez Inmaculada,Pastor Nuria,Burgos-Morón Estefanía,López-Lázaro Miguel,Cortés Felipe,Mateos Santiago,Helleday Thomas

Publisher

Oxford University Press (OUP)

Subject

Genetics

Link

http://academic.oup.com/nar/article-pdf/41/11/5827/16953696/gkt270.pdf

Reference44 articles.

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3. DNA methylation inhibitor 5-aza-2′-deoxycytidine induces reversible genome-wide DNA damage that is distinctly influenced by DNA methyltransferases 1 and 3B;Palii;Mol. Cell. Biol.,2008

4. Toxicity of 5-aza-2′-deoxycytidine to mammalian cells is mediated primarily by covalent trapping of DNA methyltransferase rather than DNA demethylation;Juttermann;Proc. Natl Acad. Sci. USA,1994

5. Trapped in action: direct visualization of DNA methyltransferase activity in living cells;Schermelleh;Nat. Methods,2005

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