DNA binding capacity of the WT1 protein is abolished by Denys—Drash syndrome WT1 point mutations-Reference-Cited by-同舟云学术

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DNA binding capacity of the WT1 protein is abolished by Denys—Drash syndrome WT1 point mutations

Published:1995 Issue:3 Volume:4 Page:351-358
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Hum Mol Genet

Author:

Little Melissa,Holmes Gregory,Bickmore Wendy,van Heyningen Veronica,Hastie Nicholas,Wainwright Brandon

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/4/3/351/1585767/4-3-351.pdf

Cited by 96 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Single‐cell transcriptomics identifies aberrant glomerular angiogenic signalling in the early stages of WT1 kidney disease;The Journal of Pathology;2024-08-23

2. A review of the genetic background in complicated WT1-related disorders;Clinical and Experimental Nephrology;2024-07-13

3. Evaluating Established Roles, Future Perspectives and Methodological Heterogeneity for Wilms’ Tumor 1 (WT1) Antigen Detection in Adult Renal Cell Carcinoma, Using a Novel N-Terminus Targeted Antibody (Clone WT49);Biomedicines;2022-04-15

4. New mutation in WT1 gene in a boy with an incomplete form of Denys-Drash syndrome;Medicine;2021-05-14

5. Case Report: Denys–Drash Syndrome With WT1 Causative Variant Presenting as Atypical Hemolytic Uremic Syndrome;Frontiers in Pediatrics;2020-12-18

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