MTHFR Polymorphism, Methyl-Replete Diets and the Risk of Colorectal Carcinoma and Adenoma among U.S. Men and Women: An Example of Gene-Environment Interactions in Colorectal Tumorigenesis-Reference-Cited by-同舟云学术

MTHFR Polymorphism, Methyl-Replete Diets and the Risk of Colorectal Carcinoma and Adenoma among U.S. Men and Women: An Example of Gene-Environment Interactions in Colorectal Tumorigenesis

Published:1999-02-01 Issue:2 Volume:129 Page:560S-564S
ISSN:0022-3166
Container-title:The Journal of Nutrition
language:en
Short-container-title:

Author:

Chen Jia¹²,Giovannucci Edward L.³²,Hunter David J.¹⁴²

Affiliation:

1. Department of Epidemiology, Harvard Medical School, Boston, MA 02115

2. Channing Laboratory, Department of Medicine, Brigham and Woman's Hospital, Harvard Medical School, Boston, MA 02115

3. Department of Nutrition, Harvard School of Public Health, Harvard Medical School, Boston, MA 02115

4. Department of The Harvard Center for Cancer Prevention, Harvard Medical School, Boston, MA 02115

Publisher

Oxford University Press (OUP)

Subject

Nutrition and Dietetics,Medicine (miscellaneous)

Link

http://academic.oup.com/jn/article-pdf/129/2/560S/24023236/4w02x90s560.pdf

Reference30 articles.

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3. Nutritional factors in colorectal cancer risk: a case-control study in Majorca;Benito;Int. J. Cancer,1991

4. Folate dificiency causes uracil misincorporation into human DNA and chromosome breakage: implications for cancer and neuronal damage;Blount;Proc. Natl. Acad. Sci. U.S.A.,1997

5. A prospective study of methylenetetrahydrofolate reductase and methionine synthase gene polymorphism and risk of colorectal adenoma;Chen,1998

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