Potential Contributions of the Tobacco Nicotine-Derived Nitrosamine Ketone (NNK) in the Pathogenesis of Steatohepatitis in a Chronic Plus Binge Rat Model of Alcoholic Liver Disease-Reference-Cited by-同舟云学术

Potential Contributions of the Tobacco Nicotine-Derived Nitrosamine Ketone (NNK) in the Pathogenesis of Steatohepatitis in a Chronic Plus Binge Rat Model of Alcoholic Liver Disease

Published:2015-01-24 Issue:2 Volume:50 Page:118-131
ISSN:0735-0414
Container-title:Alcohol and Alcoholism
language:en
Short-container-title:Alcohol and Alcoholism

Author:

Zabala V.,Tong M.,Yu R.,Ramirez T.,Yalcin E. B.,Balbo S.,Silbermann E.,Deochand C.,Nunez K.,Hecht S.,de la Monte S. M.

Publisher

Oxford University Press (OUP)

Subject

General Medicine

Link

http://academic.oup.com/alcalc/article-pdf/50/2/118/17151789/agu083.pdf

Reference116 articles.

1. An LC–MS/MS method for concurrent determination of nicotine metabolites and the role of CYP2A6 in nicotine metabolite-mediated oxidative stress in SVGA astrocytes

2. Induction of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) activation in rat lung microsomes by chronic ethanol consumption and repeated running exercise

3. Purification and characterization of oxidoreductases-catalyzing carbonyl reduction of the tobacco-specific nitrosamine 4-methylnitrosamino-1-(3-pyridyl)-1-butanone (NNK) in human liver cytosol

4. CYP2E1 induced by ethanol causes oxidative stress, proteasome inhibition and cytokeratin aggresome (Mallory body-like) formation

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