The origins and sequelae of abnormal neuroendocrine function in polycystic ovary syndrome
Author:
Publisher
Oxford University Press (OUP)
Subject
Obstetrics and Gynecology,Reproductive Medicine
Link
http://academic.oup.com/humupd/article-pdf/12/4/351/6442271/dml017.pdf
Reference135 articles.
1. Androgen excess fetal programming of female reproduction: a developmental aetiology for polycystic ovary syndrome?
2. Polycystic Ovarian Morphology with Regular Ovulatory Cycles: Insights into the Pathophysiology of Polycystic Ovarian Syndrome
3. Insulin Enhancement of Luteinizing Hormone and Follicle-Stimulating Hormone Release by Cultured Pituitary Cells*
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2. Effects of inulin on intestinal flora and metabolism-related indicators in obese polycystic ovary syndrome patients;European Journal of Medical Research;2024-08-31
3. Targeted inhibition of kisspeptin neurons reverses hyperandrogenemia and abnormal hyperactive LH secretion in a preclinical mouse model of polycystic ovary syndrome;Human Reproduction;2024-07-09
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5. Brain-specificGata4downregulation inGreywickfemale mice models the metabolic subtype of polycystic ovary syndrome;2024-05-15
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