Changes in fibroblast growth factor 23 during treatment of secondary hyperparathyroidism with alfacalcidol or paricalcitol
Author:
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/27/6/2263/6906701/gfr668.pdf
Reference33 articles.
1. Regulation of Fibroblast Growth Factor-23 Signaling by Klotho
2. Human Fibroblast Growth Factor-23 Mutants Suppress Na+-dependent Phosphate Co-transport Activity and 1α,25-Dihydroxyvitamin D3 Production
3. FGF-23 Is a Potent Regulator of Vitamin D Metabolism and Phosphate Homeostasis
4. Circulating concentration of FGF-23 increases as renal function declines in patients with chronic kidney disease, but does not change in response to variation in phosphate intake in healthy volunteers
5. FGF-23 in patients with end-stage renal disease on hemodialysis
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1. Paricalcitol and Extended-Release Calcifediol for Treatment of Secondary Hyperparathyroidism in Non-Dialysis Chronic Kidney Disease: Results From a Network Meta-Analysis;The Journal of Clinical Endocrinology & Metabolism;2023-05-26
2. Associations of time-dependent changes in phosphorus levels with cardiovascular diseases in patients undergoing hemodialysis: results from the Japan Dialysis Active Vitamin D (J-DAVID) randomized clinical trial;Clinical Kidney Journal;2022-08-03
3. Selective vitamin D receptor activator Paricalcitol and its potential benefits in hemodialysis patients with secondary hyperparathyroidism;Meditsinskiy sovet = Medical Council;2022-06-19
4. The effect of vitamin D supplementation on serum levels of fibroblast growth factor- 23: A systematic review and meta-analysis of randomized controlled trials;The Journal of Steroid Biochemistry and Molecular Biology;2022-01
5. The effect of vitamin D supplementation on fibroblast growth factor‐23 in patients with chronic kidney disease: A systematic review and meta‐analysis;Phytotherapy Research;2021-04-30
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