Anthracycline cardiotoxicity: looking for new therapeutic approaches targeting cell senescence?
Author:
Affiliation:
1. INSERM U955 and Département de Physiologie, Hôpital Henri Mondor, AP-HP, DHU A-TVB, 94010 Créteil, France
2. Université Paris-Est Créteil (UPEC), Creteil, France
Publisher
Oxford University Press (OUP)
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology
Link
http://academic.oup.com/cardiovascres/article-pdf/114/10/1304/25195521/cvy108.pdf
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3. Co-administration of resveratrol with doxorubicin in young mice attenuates detrimental late-occurring cardiovascular changes;Matsumura;Cardiovasc Res,2018
4. Juvenile exposure to anthracyclines impairs cardiac progenitor cell function and vascularization resulting in greater susceptibility to stress-induced myocardial injury in adult mice;Huang;Circulation,2010
5. Doxorubicin targets multiple players: a new view of an old problem;Cappetta;Pharmacol Res,2018
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1. Stress-induced premature senescence is associated with a prolonged QT interval and recapitulates features of cardiac aging;Theranostics;2022
2. The Interplay Between Autophagy and Senescence in Anthracycline Cardiotoxicity;Current Heart Failure Reports;2021-06-03
3. Possibilities of preclinical diagnosis of anthracycline cardiotoxicity using the technique of "speckle-tracking echocardiography";Bulletin of the Russian Military Medical Academy;2021-05-12
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