Vacuolar uptake of host components, and a role for cholesterol and sphingomyelin in malarial infection-Reference-Cited by-同舟云学术

Vacuolar uptake of host components, and a role for cholesterol and sphingomyelin in malarial infection

Author:

Lauer Sabine¹²,VanWye Jeffrey¹,Harrison Travis³,McManus Heather³,Samuel Benjamin U.³,Hiller N.Luisa³,Mohandas Narla⁴,Haldar Kasturi³

Affiliation:

1. Department of Microbiology and Immunology, Stanford University School of Medicine; Stanford CA 94305 USA

2. Present address: Los Alamos National Laboratory, Health Research Laboratory (HRL 1), MS 888, LS-5; Los Alamos NM 87545 USA

3. Departments of Pathology and Microbiology-Immunology, Northwestern University; 303 E Chicago Avenue Chicago IL 60611 USA

4. Life Science Division, Lawrence Berkeley National Laboratory; 1 Cyclotron Road Berkeley CA 94720 USA

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference39 articles.

1. The caveolae membrane system;Anderson;Annu Rev Biochem,1998

2. Vesicle-mediated transport of membrane and proteins in malaria-infected erythrocytes;Barnwell;Blood Cells,1990

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