NOBOX is a key FOXL2 partner involved in ovarian folliculogenesis
Author:
Publisher
Oxford University Press (OUP)
Subject
Cell Biology,Genetics,Molecular Biology,General Medicine
Link
http://academic.oup.com/jmcb/article-pdf/6/2/175/14098470/mju006.pdf
Reference10 articles.
1. The identification and characterization of a FOXL2 response element provides insights into the pathogenesis of mutant alleles;Benayoun;Hum. Mol. Genet.,2008
2. FOXL2 mutations and genomic rearrangements in BPES;Beysen;Hum. Mutat.,2009
3. Novel NOBOX loss-of-function mutations account for 6.2% of cases in a large primary ovarian insufficiency cohort;Bouilly;Hum. Mutat.,2011
4. Characterization of NOBOX DNA binding specificity and its regulation of Gdf9 and Pou5f1 promoters;Choi;J. Biol. Chem.,2006
5. Joint regulation of the MAP1B promoter by HNF3beta/Foxa2 and Engrailed is the result of a highly conserved mechanism for direct interaction of homeoproteins and Fox transcription factors;Foucher;Development,2003
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