Imatinib ameliorates fibrosis in uraemic cardiac disease in BALB/c without improving cardiac function-Reference-Cited by-同舟云学术

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Imatinib ameliorates fibrosis in uraemic cardiac disease in BALB/c without improving cardiac function

Published:2010-01-07 Issue:6 Volume:25 Page:1817-1824
ISSN:0931-0509
Container-title:Nephrology Dialysis Transplantation
language:en
Short-container-title:Nephrology Dialysis Transplantation

Author:

Baumann M.,Leineweber K.,Tewiele M.,Wu K.,Turk T. R.,Su S.,Gossl M.,Buck T.,Wilde B.,Heemann U.,Kribben A.,Witzke O.

Publisher

Oxford University Press (OUP)

Subject

Transplantation,Nephrology

Link

http://academic.oup.com/ndt/article-pdf/25/6/1817/5339386/gfp708.pdf

Reference30 articles.

1. Morphology of the heart and arteries in renal failure

2. Structural Causes of Cardiac Dysfunction in Uremia

3. Electrocardiographic abnormalities and uremic cardiomyopathy

4. Platelet-Derived Growth Factor D Induces Cardiac Fibrosis and Proliferation of Vascular Smooth Muscle Cells in Heart-Specific Transgenic Mice

5. Structural and functional specificities of PDGF-C and PDGF-D, the novel members of the platelet-derived growth factors family

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1. In Humanized Sickle Cell Mice, Imatinib Protects Against Sickle Cell–Related Injury;HemaSphere;2023-02-28

2. Cardiac Remodeling in Chronic Kidney Disease;Toxins;2020-03-05

3. Chronic kidney disease induces a systemic microangiopathy, tissue hypoxia and dysfunctional angiogenesis;Scientific Reports;2018-03-28

4. Diosgenin, a steroidal saponin, prevents hypertension, cardiac remodeling and oxidative stress in adenine induced chronic renal failure rats;RSC Advances;2015

5. Target organ cross talk in cardiorenal syndrome: animal models;American Journal of Physiology-Renal Physiology;2012-11-01

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