Depletion of extracellular signal-regulated kinase 1 in mice with cardiomyopathy caused by lamin A/C gene mutation partially prevents pathology before isoenzyme activation-Reference-Cited by-同舟云学术

Depletion of extracellular signal-regulated kinase 1 in mice with cardiomyopathy caused by lamin A/C gene mutation partially prevents pathology before isoenzyme activation

Published:2013-08-09 Issue:1 Volume:23 Page:1-11
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Wu W.,Iwata S.,Homma S.,Worman H. J.,Muchir A.

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/23/1/1/2136955/ddt387.pdf

Reference43 articles.

1. The Nuclear Envelope as a Signaling Node in Development and Disease

2. Laminopathies and the long strange trip from basic cell biology to therapy

3. LMNA cardiomyopathy: cell biology and genetics meet clinical medicine

4. Mutations in the gene encoding lamin A/C cause autosomal dominant Emery-Dreifuss muscular dystrophy

5. Missense Mutations in the Rod Domain of the Lamin A/C Gene as Causes of Dilated Cardiomyopathy and Conduction-System Disease

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