Maternal insulin resistance causes oxidative stress and mitochondrial dysfunction in mouse oocytes-Reference-Cited by-同舟云学术

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Maternal insulin resistance causes oxidative stress and mitochondrial dysfunction in mouse oocytes

Published:2012-05-03 Issue:7 Volume:27 Page:2130-2145
ISSN:0268-1161
Container-title:Human Reproduction
language:en
Short-container-title:Human Reproduction

Author:

Ou X.-H.,Li S.,Wang Z.-B.,Li M.,Quan S.,Xing F.,Guo L.,Chao S.-B.,Chen Z.,Liang X.-W.,Hou Y.,Schatten H.,Sun Q.-Y.

Publisher

Oxford University Press (OUP)

Subject

Obstetrics and Gynecology,Rehabilitation,Reproductive Medicine

Link

http://academic.oup.com/humrep/article-pdf/27/7/2130/2389516/des137.pdf

Reference74 articles.

1. Insulin Signaling in Mouse Oocytes1

2. Oxidative stress and its implications in female infertility – a clinician's perspective

3. Redox Considerations in Female Reproductive Function and Assisted Reproduction: From Molecular Mechanisms to Health Implications

4. Lack of maternal Heat Shock Factor 1 results in multiple cellular and developmental defects, including mitochondrial damage and altered redox homeostasis, and leads to reduced survival of mammalian oocytes and embryos

5. Insulin regulation of mitochondrial proteins and oxidative phosphorylation in human muscle

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