Suppression of inflammatory and neuropathic pain symptoms in mice lacking the N-type Ca2+ channel
Author:
Publisher
Wiley
Subject
General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience
Cited by 307 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Voltage-dependent G-protein regulation of Ca V 2.2 (N-type) channels;Science Advances;2024-09-13
2. Uncoupling the CRMP2-CaV2.2 interaction reduces pain-like behavior in a preclinical joint-pain model;The Journal of Pain;2024-09
3. Electrical and G-protein Regulation of CaV2.2 (N-type) Channels;2024-06-30
4. Uncoupling the CRMP2-CaV2.2 interaction reduces pain-like behavior in a preclinical osteoarthritis model;2024-06-06
5. Interleukin-1α links peripheral CaV2.2 channel activation to rapid adaptive increases in heat sensitivity in skin;Scientific Reports;2024-04-20
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