Direct detection in clinical samples of multiple gene mutations causing resistance of Mycobacterium tuberculosis to isoniazid and rifampicin using fluorogenic probes

Author:

Espasa Mateu,González-Martín Julián,Alcaide Fernando,Aragón Lina Marcela,Lonca Joan,Manterola Joxe Mari,Salvadó Margarita,Tudó Griselda,Orús Pilar,Coll Pere

Publisher

Oxford University Press (OUP)

Subject

Infectious Diseases,Pharmacology (medical),Pharmacology,Microbiology (medical)

Reference32 articles.

1. WHO. Global Tuberculosis Control. Surveillance, Planning, Financing. WHO/CDS/TB2003.316. Geneva, Switzerland: WHO, 2003.

2. Espinal MA, Laszlo A, Simonsen L et al. Global trends in resistance to antituberculosis drugs. N Engl J Med2001; 344: 1294–303.

3. Metchock BG, Nolte FS, Wallace RJ, Jr. Mycobacterium. In: Murray PR, Baron EJ, Pfaller MA et al., ed. Manual of Clinical Microbiology. Washington, DC, USA: American Society for Microbiology, 1999; 399–437.

4. García de Viedma D. Rapid detection of resistance in Mycobacterium tuberculosis: a review discussing molecular approaches. Clin Microbiol Infect2003; 9: 349–59.

5. Mdluli K, Slayden RA, Zhu Y et al. Inhibition of a Mycobacterium tuberculosis β-ketoacyl ACP synthase by isoniazid. Science1998; 280: 1607–10.

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