Sarcolipin deletion in mdx mice impairs calcineurin signalling and worsens dystrophic pathology
Author:
Affiliation:
1. Department of Kinesiology, University of Waterloo, Waterloo, ON N2L 3G1 Canada
Funder
Canadian Institutes of Health Research
Publisher
Oxford University Press (OUP)
Subject
Genetics (clinical),Genetics,Molecular Biology,General Medicine
Link
http://academic.oup.com/hmg/advance-article-pdf/doi/10.1093/hmg/ddy302/25682203/ddy302.pdf
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3. Calcineurin-NFAT signaling, together with GABP and peroxisome PGC-1{alpha}, drives utrophin gene expression at the neuromuscular junction;Angus;Am. J. Physiol. Cell Physiol.,2005
4. Stimulation of calcineurin signaling attenuates the dystrophic pathology in mdx mice;Chakkalakal;Hum. Mol. Genet.,2004
5. The functional role of calcineurin in hypertrophy, regeneration, and disorders of skeletal muscle;Sakuma;J. Biomed. Biotechnol.,2010
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