A severe mouse model of spinal muscular atrophy develops early systemic inflammation-Reference-Cited by-同舟云学术

A severe mouse model of spinal muscular atrophy develops early systemic inflammation

Published:2018-09-04 Issue: Volume: Page:
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Wan Bo¹²,Feng Pengchao¹²,Guan Zeyuan¹²,Sheng Lei¹²,Liu Zhiyong³⁴,Hua Yimin¹²

Affiliation:

1. Jiangsu Key Laboratory of Neuropsychiatric Diseases, Department of Neurology and Suzhou Clinical Research Center of Neurological Disease, Hospital of Soochow University, Suzhou, Jiangsu, China

2. Institute of Neuroscience, Soochow University, Suzhou, Jiangsu, China

3. School for Radiological and Interdisciplinary Sciences (RAD-X), Soochow University, Suzhou, Jiangsu, China

4. Collaborative Innovation Center of Radiation Medicine of Jiangsu Higher Education Institutions, Soochow University, Suzhou, Jiangsu, China

Funder

National Natural Science Foundation of China

Suzhou Clinical Research Center of Neurological Disease

Priority Academic Program Development of Jiangsu Higher Education Institutions

Jiangsu Key Laboratory of Neuropsychiatric Diseases

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/advance-article-pdf/doi/10.1093/hmg/ddy300/25682713/ddy300.pdf

Reference83 articles.

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3. A single nucleotide in the SMN gene regulates splicing and is responsible for spinal muscular atrophy;Lorson;Proc. Natl. Acad. Sci. U. S. A.,1999

4. A-44G transition in SMN2 intron 6 protects patients with spinal muscular atrophy;Wu;Hum. Mol. Genet.,2017

5. The spinal muscular atrophy disease gene product, SMN: A link between snRNP biogenesis and the Cajal (coiled) body;Carvalho;J. Cell Biol.,1999

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