Expression of wild-type human superoxide dismutase-1 in mice causes amyotrophic lateral sclerosis
Author:
Publisher
Oxford University Press (OUP)
Subject
Genetics (clinical),Genetics,Molecular Biology,General Medicine
Link
http://academic.oup.com/hmg/article-pdf/22/1/51/1697403/dds399.pdf
Reference50 articles.
1. Clinical genetics of amyotrophic lateral sclerosis: what do we really know?
2. Neurodegenerative diseases: a decade of discoveries paves the way for therapeutic breakthroughs
3. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis withSOD1 mutations
4. New consensus research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 (SOD1) gene mutations: Inclusions containing SOD1 in neurons and astrocytes
5. Aggregation and Motor Neuron Toxicity of an ALS-Linked SOD1 Mutant Independent from Wild-Type SOD1
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