Mutant spastin proteins promote deficits in axonal transport through an isoform-specific mechanism involving casein kinase 2 activation-Reference-Cited by-同舟云学术

Mutant spastin proteins promote deficits in axonal transport through an isoform-specific mechanism involving casein kinase 2 activation

Author:

Leo Lanfranco¹,Weissmann Carina²,Burns Matthew²,Kang Minsu²³,Song Yuyu³⁴,Qiang Liang¹,Brady Scott T.²³,Baas Peter W.¹,Morfini Gerardo²³

Affiliation:

1. Department of Neurobiology and Anatomy, Drexel University College of Medicine, Philadelphia, PA, USA

2. Department of Anatomy and Cell Biology, University of Illinois at Chicago, Chicago, IL, USA

3. Marine Biological Laboratory, Woods Hole, MA, USA

4. Department of Genetics, School of Medicine, Yale University, New Haven, CT, USA

Funder

National Institutes of Health

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

Reference90 articles.

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3. Hereditary spastic paraplegia SPG4: what is known and not known about the disease;Solowska;Brain,2015

4. Hereditary spastic paraplegia: clinico-pathologic features and emerging molecular mechanisms;Fink;Acta Neuropathol,2013

5. Spastin, the protein mutated in autosomal dominant hereditary spastic paraplegia, is involved in microtubule dynamics;Errico;Hum. Mol. Genet,2002

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