BRAF and EGFR inhibitors synergize to increase cytotoxic effects and decrease stem cell capacities in BRAF(V600E)-mutant colorectal cancer cells
Author:
Affiliation:
1. Department of Medical Oncology, Fudan University Shanghai Cancer Center, Shanghai 200032, China
2. Department of Oncology, Shanghai Medical College, Fudan University, Shanghai 200032, China
Funder
National Natural Science Foundation of China
Shanghai Municipal Commission of Health and Family Planning
Publisher
China Science Publishing & Media Ltd.
Subject
General Medicine,Biochemistry,Biophysics
Link
http://academic.oup.com/abbs/article-pdf/50/4/355/24593796/gmy018.pdf
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1. Role and Function of Receptor Tyrosine Kinases in BRAF Mutant Cancers;Receptors;2024-03-04
2. Colorectal Cancer Stem Cells and Targeted Agents;Pharmaceutics;2023-12-12
3. Chalcone/1,3,4-Oxadiazole/Benzimidazole hybrids as novel anti-proliferative agents inducing apoptosis and inhibiting EGFR & BRAFV600E;BMC Chemistry;2023-09-16
4. Targeted Therapies in Colorectal Cancer: Recent Advances in Biomarkers, Landmark Trials, and Future Perspectives;Cancers;2023-06-01
5. BRAF gene as a potential target to attenuate drug resistance and treat cancer;Gene Reports;2023-03
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