TNFAIP3 gene polymorphisms associated with differential susceptibility to rheumatoid arthritis and systemic lupus erythematosus in the Korean population-Reference-Cited by-同舟云学术

TNFAIP3 gene polymorphisms associated with differential susceptibility to rheumatoid arthritis and systemic lupus erythematosus in the Korean population

Published:2014-01-31 Issue:6 Volume:53 Page:1009-1013
ISSN:1462-0324
Container-title:Rheumatology
language:en
Short-container-title:Rheumatology

Author:

Kim S.-K.,Choe J.-Y.,Bae J.,Chae S.-C.,Park D.-J.,Kwak S. G.,Lee S.-S.

Publisher

Oxford University Press (OUP)

Subject

Pharmacology (medical),Rheumatology

Link

http://academic.oup.com/rheumatology/article-pdf/53/6/1009/5167704/ket473.pdf

Reference20 articles.

1. The ubiquitin-modifying enzyme A20 is required for termination of Toll-like receptor responses

2. Failure to Regulate TNF-Induced NF-κB and Cell Death Responses in A20-Deficient Mice

3. The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology

4. Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

5. Two independent alleles at 6q23 associated with risk of rheumatoid arthritis

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3. Association of TNFAIP3 gene polymorphism (rs5029939) with susceptibility and clinical phenotype of systemic lupus erythematosus;ARCH RHEUMATOL;2021

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