A single nucleotide polymorphism in exon 1 of cytotoxic T-lymphocyte-associated-4 (CTLA-4) is not associated with rheumatoid arthritis

Author:

Barton A.,Myerscough A.,John S.,Gonzalez-Gay M.,Ollier W.,Worthington J.

Publisher

Oxford University Press (OUP)

Subject

Pharmacology (medical),Rheumatology

Reference14 articles.

1. Silman AJ, MacGregor AJ, Thomson W et al. Twin concordance rates for rheumatoid arthritis: results from a nationwide study. Br J Rheumatol1993;32:903–7.

2. MacGregor AJ, Rigby AS, Ollier WER, Silman AJ. An estimation of the relative genetic and environmental contribution to rheumatoid arthritis based on covariance analysis of twin data. Br J Rheumatol1994;33(suppl. 1):1583.

3. John S, Hajeer A, Marlow A et al. Investigation of candidate disease susceptibility genes in rheumatoid arthritis: principles and strategies. J Rheumatol1997;24: 199–201.

4. Waterhouse P, Penninger JM, Timms E et al. Lymphoproliferative disorders with early lethality in mice deficient in CTLA-4. Science1995;270:985–8.

5. Nistico L, Buzzetti R, Pritchard L et al. The CTLA-4 gene region of chromosome 2q33 is linked to and associated with type I diabetes. Hum Mol Genet1996;5:1075–86.

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