A longitudinal cohort study of the anti-synthetase syndrome: increased severity of interstitial lung disease in black patients and patients with anti-PL7 and anti-PL12 autoantibodies

Author:

Pinal-Fernandez Iago1,Casal-Dominguez Maria2,Huapaya Julio A.2,Albayda Jemima2,Paik Julie J.2,Johnson Cheilonda2,Silhan Leann2,Christopher-Stine Lisa2,Mammen Andrew L.1,Danoff Sonye K.2

Affiliation:

1. National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda

2. Johns Hopkins University School of Medicine, Baltimore, MD, USA

Funder

National Institutes of Health

National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Oxford University Press (OUP)

Subject

Pharmacology (medical),Rheumatology

Reference26 articles.

1. Heterogeneity of precipitating antibodies in polymyositis and dermatomyositis. Characterization of the Jo-1 antibody system;Nishikai;Arthritis Rheum,1980

2. Anti-threonyl-tRNA synthetase, a second myositis-related autoantibody;Mathews;J Exp Med,1984

3. Autoantibodies against alanyl-tRNA synthetase and tRNAAla coexist and are associated with myositis;Bunn;J Exp Med,1986

4. Autoantibodies to aminoacyl-transfer RNA synthetases for isoleucine and glycine. Two additional synthetases are antigenic in myositis;Targoff;J Immunol,1990

5. Anti-KS: identification of autoantibodies to asparaginyl-transfer RNA synthetase associated with interstitial lung disease;Hirakata;J Immunol,1999

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