Advanced glycation end products cause collagen II reduction by activating Janus kinase/signal transducer and activator of transcription 3 pathway in porcine chondrocytes-Reference-Cited by-同舟云学术

Advanced glycation end products cause collagen II reduction by activating Janus kinase/signal transducer and activator of transcription 3 pathway in porcine chondrocytes

Published:2011-04-10 Issue:8 Volume:50 Page:1379-1389
ISSN:1462-0324
Container-title:Rheumatology
language:en
Short-container-title:Rheumatology

Author:

Huang C.-Y.,Lai K.-Y.,Hung L.-F.,Wu W.-L.,Liu F.-C.,Ho L.-J.

Publisher

Oxford University Press (OUP)

Subject

Pharmacology (medical),Rheumatology

Link

http://academic.oup.com/rheumatology/article-pdf/50/8/1379/5066049/ker134.pdf

Reference35 articles.

1. Osteoarthritis

2. Risk factors for onset of osteoarthritis of the knee in older adults: a systematic review and meta-analysis

3. Articular chondrocytes express the receptor for advanced glycation end products: Potential role in osteoarthritis

4. Accumulation of advanced glycation end products as a molecular mechanism for aging as a risk factor in osteoarthritis

5. Crosslinking by advanced glycation end products increases the stiffness of the collagen network in human articular cartilage: A possible mechanism through which age is a risk factor for osteoarthritis

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