PTPN22 R620W genotype-phenotype correlation analysis and gene-environment interaction study in early rheumatoid arthritis: results from the ESPOIR cohort-Reference-Cited by-同舟云学术

PTPN22 R620W genotype-phenotype correlation analysis and gene-environment interaction study in early rheumatoid arthritis: results from the ESPOIR cohort

Published:2011-07-13 Issue:10 Volume:50 Page:1802-1808
ISSN:1462-0324
Container-title:Rheumatology
language:en
Short-container-title:Rheumatology

Author:

Salliot C.,Dawidowicz K.,Lukas C.,Guedj M.,Paccard C.,Benessiano J.,Dougados M.,Nicaise P.,Meyer O.,Dieude P.

Publisher

Oxford University Press (OUP)

Subject

Pharmacology (medical),Rheumatology

Link

http://academic.oup.com/rheumatology/article-pdf/50/10/1802/5066623/ker224.pdf

Reference26 articles.

1. A new model for an etiology of rheumatoid arthritis: Smoking may trigger HLA–DR (shared epitope)–restricted immune reactions to autoantigens modified by citrullination

2. Gene–environment interaction between HLA-DRB1 shared epitope and heavy cigarette smoking in predicting incident rheumatoid arthritis

3. The PTPN22 C1858T functional polymorphism and autoimmune diseases--a meta-analysis

4. Risk of progression from undifferentiated arthritis to rheumatoid arthritis: the effect of the PTPN22 1858T-allele in anti-citrullinated peptide antibody positive patients

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