Contribution of PTPN22 1858T, TNFRII 196R and HLA-shared epitope alleles with rheumatoid factor and anti-citrullinated protein antibodies to very early rheumatoid arthritis diagnosis-Reference-Cited by-同舟云学术

Contribution of PTPN22 1858T, TNFRII 196R and HLA-shared epitope alleles with rheumatoid factor and anti-citrullinated protein antibodies to very early rheumatoid arthritis diagnosis

Published:2008-04-29 Issue:8 Volume:47 Page:1208-1212
ISSN:1462-0324
Container-title:Rheumatology
language:en
Short-container-title:Rheumatology

Author:

Goeb V.,Dieude P.,Daveau R.,Thomas-L'Otellier M.,Jouen F.,Hau F.,Boumier P.,Tron F.,Gilbert D.,Fardellone P.,Cornelis F.,Le Loet X.,Vittecoq O.

Publisher

Oxford University Press (OUP)

Subject

Pharmacology (medical),Rheumatology

Link

http://academic.oup.com/rheumatology/article-pdf/47/8/1208/5054697/ken192.pdf

Reference34 articles.

1. Benefit of very early referral and very early therapy with disease-modifying anti-rheumatic drugs in patients with early rheumatoid arthritis

2. The shared epitope hypothesis. an approach to understanding the molecular genetics of susceptibility to rheumatoid arthritis

3. A Missense Single-Nucleotide Polymorphism in a Gene Encoding a Protein Tyrosine Phosphatase (PTPN22) Is Associated with Rheumatoid Arthritis

4. Quantifying the exact role of HLA-DRB1 alleles in susceptibility to inflammatory polyarthritis: Results from a large, population-based study

5. The genetics revolution and the assault on rheumatoid arthritis

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