Cross-species genetic screens to identify kinase targets for APP reduction in Alzheimer's disease-Reference-Cited by-同舟云学术

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Cross-species genetic screens to identify kinase targets for APP reduction in Alzheimer's disease

Published:2019-02-12 Issue:12 Volume:28 Page:2014-2029
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Huichalaf Claudia H¹,Al-Ramahi Ismael²³,Park Kyung-Won¹,Grunke Stacy D¹,Lu Nan²³,de Haro Maria²³,El-Zein Karla²³,Gallego-Flores Tatiana²³,Perez Alma M²³,Jung Sung Yun⁴,Botas Juan²⁵³,Zoghbi Huda Y¹²⁶⁷³,Jankowsky Joanna L¹⁵⁷⁸

Affiliation:

1. Department of Neuroscience

2. Department of Molecular and Human Genetics

3. Jan and Dan Duncan Neurological Research Institute, Texas Children's Hospital, Houston, TX, USA

4. Department of Biochemistry

5. Department of Molecular and Cellular Biology

6. Department of Pediatrics

7. Department of Neurology

8. Department of Neurosurgery, Baylor College of Medicine, Houston, TX, USA

Funder

CPRIT Core Facility Award

Intellectual and Developmental Disabilities Research Center

BrightFocus Foundation

National Institutes of Health

Robert A. and Renee E. Belfer Family Foundation

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/28/12/2014/28765653/ddz034.pdf

Reference62 articles.

1. The elusive nature and diagnostics of misfolded Aβ oligomers;Cerasoli;Front. Chem.,2015

2. Truncated and modified amyloid-beta species;Kummer;Alzheimers Res. Ther.,2014

3. Secretase inhibitors for the treatment of Alzheimer's disease: long road ahead;Kumar;Eur. J. Med. Chem.,2018

4. Memory deficits of British dementia knock-in mice are prevented by Aβ-precursor protein haploinsufficiency;Tamayev;J. Neurosci.,2012

5. β-Amyloid precursor protein-deficient mice show reactive gliosis and decreased locomotor activity;Zheng;Cell,1995

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