Long-term treatment with naproxcinod significantly improves skeletal and cardiac disease phenotype in the mdx mouse model of dystrophy-Reference-Cited by-同舟云学术

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Long-term treatment with naproxcinod significantly improves skeletal and cardiac disease phenotype in the mdx mouse model of dystrophy

Published:2014-01-23 Issue:12 Volume:23 Page:3239-3249
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Uaesoontrachoon K.,Quinn J. L.,Tatem K. S.,Van Der Meulen J. H.,Yu Q.,Phadke A.,Miller B. K.,Gordish-Dressman H.,Ongini E.,Miglietta D.,Nagaraju K.

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/23/12/3239/2054430/ddu033.pdf

Reference64 articles.

1. Membrane protein kinase alteration in Duchenne muscular dystrophy

2. Dystrophin and disease

3. Dystrophin: The protein product of the duchenne muscular dystrophy locus

4. Deficiency of a glycoprotein component of the dystrophin complex in dystrophic muscle

5. Dystrophin-associated proteins are greatly reduced in skeletal muscle from mdx mice.

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2. Effects of Chronic, Maximal Phosphorodiamidate Morpholino Oligomer (PMO) Dosing on Muscle Function and Dystrophin Restoration in a Mouse Model of Duchenne Muscular Dystrophy;Journal of Neuromuscular Diseases;2021-11-30

3. Cardioprotective effect of nicorandil on isoproterenol induced cardiomyopathy in the Mdx mouse model;BMC Cardiovascular Disorders;2021-06-15

4. Multi-Compartment, Early Disruption of cGMP and cAMP Signalling in Cardiac Myocytes from the mdx Model of Duchenne Muscular Dystrophy;International Journal of Molecular Sciences;2020-09-25

5. Enhanced dimethylarginine degradation improves coronary flow reserve and exercise tolerance in Duchenne muscular dystrophy carrier mice;American Journal of Physiology-Heart and Circulatory Physiology;2020-09-01

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