The histone deacetylase HDAC3 is essential for Purkinje cell function, potentially complicating the use of HDAC inhibitors in SCA1-Reference-Cited by-同舟云学术

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The histone deacetylase HDAC3 is essential for Purkinje cell function, potentially complicating the use of HDAC inhibitors in SCA1

Published:2014-03-04 Issue:14 Volume:23 Page:3733-3745
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Venkatraman Anand,Hu Yuan-Shih,Didonna Alessandro,Cvetanovic Marija,Krbanjevic Aleksandar,Bilesimo Patrice,Opal Puneet

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/23/14/3733/13917053/ddu081.pdf

Reference60 articles.

1. The Hereditary Ataxias

2. Trinucleotide repeat disorders;Orr;Annu. Rev. Neurosci.,2007

3. Polyglutamine expansion down-regulates specific neuronal genes before pathologic changes in SCA1;Lin;Nat. Neurosci.,2000

4. Gene profiling links SCA1 pathophysiology to glutamate signaling in Purkinje cells of transgenic mice;Serra;Hum. Mol. Genet.,2004

5. RORalpha-mediated Purkinje cell development determines disease severity in adult SCA1 mice;Serra;Cell,2006

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