Mass Transport Mechanism in the Retina of the Human Eye

Abstract

The metabolic secretions produced by the cells inside the human eye are accumulated simultaneously in all of its parts; subject only to their origin. However, the symptomatology is well characterized by the location of accumulation. Genetic factors are directly related to how the clumps of metabolic secretions occur and the form of accumulation is very specific to the movement of the eyes based on the activities performed by the patient, so any outcome is unpredictable. The outer retinal layers are avascular and are supplied by diffusion from the choriocapillatis. Abnormalities of retinal blood circulation and drainage of metabolic secretions are key indicators to retinal dysfunction and may point to perturbations of systemic circulation. The weakening of the cornea bending mechanism results in the loss of variation of intraocular pressure. Consequently, due to the increase of its work activities, the weakening of the forced convection mechanism of the metabolic secretions produced in the retina initiates the accumulation of these secretions without uniformity in the retina, causing gradual obstruction of its system of mass transport, nutrients and metabolic secretions. This accumulation is readily observed by pressing ones hands for several minutes on closed eyes, subsequently, there is the perception of light in one without a region of uniformity. This work analyzes the anatomic structure and biophysics, depicted in the scientific literature, along with the symptoms of the presbyopia treatment of the first author of this paper, to understand that the accumulation of metabolic secretions causes an agglutination and dehydration process by decanting that triggers the loss of flexibility of the fibrous coat, vascular coat (uveal tissue) and nervous coat (retina) and consequently the gradual obstruction of the system of mass transport in the retina, compromising its nutrition and obstructing the drainage of its metabolic secretion.