Numerical analysis of bursting activity in an isolated pancreatic β-cell model
Author:
Affiliation:
1. Division of Policy and Planning Sciences, Faculty of Engineering, Information and Systems, University of Tsukuba
2. Department of Management Science, Graduate School of Engineering, Tokyo University of Science
Publisher
Institute of Electronics, Information and Communications Engineers (IEICE)
Subject
Rehabilitation,Physical Therapy, Sports Therapy and Rehabilitation,General Medicine
Link
https://www.jstage.jst.go.jp/article/nolta/7/2/7_217/_pdf
Reference14 articles.
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2. [2] F. Martin and B. Soria B, “Glucose-induced [Ca2+]i oscillations in single human pancreatic islets,” Cell Calcium, vol. 20, pp. 409-414, 1996.
3. [3] E. Andreu, B. Soria, and J.V. Sanchez-Andres, “Oscillation of gap junction electrical coupling in the mouse pancreatic islets of Langerhans,” J. Physiol., vol. 498, pp. 753-761, 1997.
4. [4] P. Smolen, J. Rinzel, and A. Sherman, “Why pancreatic islets burst but single beta cells do not. The heterogeneity hypothesis,” Biophys. J., vol. 64, pp. 1668-1680, 1993.
5. [5] M. Yoshida, M. Nakata, S. Yamato, K. Dezaki, H. Sugawara, S. Ishikawa, M. Kawakami, T. Yada, and M. Kakei, “Voltage-dependent metabolic regulation of Kv2.1 channels in pancreatic β-cells,” Biochem. Biophys. Res. Commun., vol. 396, pp. 304-309, 2010.
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