Hematopoietic MyD88 and IL-18 are essential for IFN-γ–dependent restriction of type A Francisella tularensis infection
Author:
Publisher
Wiley
Subject
Cell Biology,Immunology,Immunology and Allergy
Cited by 7 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Francisella tularensis disrupts TLR2-MYD88-p38 signaling early during infection to delay apoptosis of macrophages and promote virulence in the host;mBio;2023-07-05
2. MyD88-Dependent Glucose Restriction and Itaconate Production Control Brucella Infection;Infection and Immunity;2021-09-16
3. Modification of the Pulmonary MyD88 Inflammatory Response Underlies the Role of the Yersinia pestis Pigmentation Locus in Primary Pneumonic Plague;Infection and Immunity;2021-02-16
4. Production of IFN-γ by splenic dendritic cells during innate immune responses against Francisella tularensis LVS depends on MyD88, but not TLR2, TLR4, or TLR9;PLOS ONE;2020-08-03
5. Innate Immune Recognition: An Issue More Complex Than Expected;Frontiers in Cellular and Infection Microbiology;2019-07-03
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