Short tail with skin lesion phenotype occurs in transgenic mice with keratin-14 promoter-directed expression of mutant CXCR2-Reference-Cited by-同舟云学术

Short tail with skin lesion phenotype occurs in transgenic mice with keratin-14 promoter-directed expression of mutant CXCR2

Published:2008-05-27 Issue:2 Volume:84 Page:406-419
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:Journal of Leukocyte Biology

Author:

Yu Yingchun,Su Yingjun,Opalenik Susan R.,Sobolik-Delmaire Tammy,Neel Nicole F.,Zaja-Milatovic Snjezana,Short Sarah T.,Sai Jiqing,Richmond Ann

Funder

Rockefeller University

Vanderbilt University

School of Medicine, Vanderbilt University

University of Virginia

University of Nebraska-Lincoln

National Institutes of Health

Skin Disease Research Center

Vanderbilt Ingram Cancer Center

U.S. Department of Veterans Affairs

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Reference47 articles.

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2. Chemokine and chemokine receptor expression in keloid and normal fibroblasts;Nirodi;Wound Repair Regen.,2000

3. Essential involvement of IL-6 in the skin wound-healing process as evidenced by delayed wound healing in IL-6-deficient mice;Lin;J. Leukoc. Biol.,2003

4. Impaired healing of nitrogen mustard wounds in CXCR2 null mice;Milatovic;Wound Repair Regen.,2003

5. Delayed wound healing and disorganized neovascularization in transgenic mice expressing the IP-10 chemokine;Luster;Proc. Assoc. Am. Physicians,1998

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1. Chapter 15 Characterization of Chemokine Receptor CXCR2 Interacting Proteins Using a Proteomics Approach to Define the CXCR2 “Chemosynapse”;Methods in Enzymology;2009