Editorial: Leishmania survival mechanisms: the role of host phosphatases
Author:
Publisher
Wiley
Subject
Cell Biology,Immunology,Immunology and Allergy
Reference11 articles.
1. Subversion mechanisms by which Leishmania parasites can escape the host immune response: a signaling point of view;Olivier;Clin. Microbiol. Rev.,2005
2. Infection with arginase-deficient Leishmania major reveals a parasite number-dependent and cytokine-independent regulation of host cellular arginase activity and disease pathogenesis;Muleme;J. Immunol.,2009
3. Large-scale structural analysis of the classical human protein tyrosine phosphatome;Barr;Cell,2009
4. Inhibition of a p38/stress-activated protein kinase-2-dependent phosphatase restores function of IL-1 receptor-associate kinase-1 and reverses Toll-like receptor 2- and 4-dependent tolerance of macrophages;Ropert;J. Immunol.,2003
5. Protein phosphatase 2A: a definite player in viral and parasitic regulation;Garcia;Microbes Infect.,2000
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