Fas (CD95) induces macrophage proinflammatory chemokine production via a MyD88-dependent, caspase-independent pathway

Author:

Altemeier William A.,Zhu Xiaodong,Berrington William R.,Harlan John M.,Liles W. Conrad

Funder

National Institutes of Health

American Lung Association Biomedical Research

Canada Research Chair in Inflammation and Infectious Diseases

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Reference39 articles.

1. Fas expression in pulmonary alveolar type II cells;Fine;Am. J. Physiol.,1997

2. Live and let die: regulatory mechanisms in Fas-mediated apoptosis;Curtin;Cell. Signal.,2003

3. Fas-mediated acute lung injury requires fas expression on nonmyeloid cells of the lung;Matute-Bello;J. Immunol.,2005

4. Recombinant human Fas ligand induces alveolar epithelial cell apoptosis and lung injury in rabbits;Matute-Bello;Am. J. Physiol. Lung Cell. Mol. Physiol.,2001

5. Fas (CD95) induces alveolar epithelial cell apoptosis in vivo: implications for acute pulmonary inflammation;Matute-Bello;Am. J. Pathol.,2001

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