Complement protein C1q bound to apoptotic cells suppresses human macrophage and dendritic cell-mediated Th17 and Th1 T cell subset proliferation-Reference-Cited by-同舟云学术

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Complement protein C1q bound to apoptotic cells suppresses human macrophage and dendritic cell-mediated Th17 and Th1 T cell subset proliferation

Published:2014-11-07 Issue:1 Volume:97 Page:147-160
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:Journal of Leukocyte Biology

Author:

Clarke Elizabeth V.,Weist Brian M.,Walsh Craig M.,Tenner Andrea J.

Funder

National Institutes of Health

Lupus Foundation of America Gina M. Finzi fellowship

National Center for Research Resources

National Center for Advancing Translational Sciences

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Reference84 articles.

1. C1q and systemic lupus erythematosus;Walport;Immunobiology,1998

2. Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies;Botto;Nat. Genet.,1998

3. C1q deficiency and autoimmunity: the effects of genetic background on disease expression;Mitchell;J. Immunol.,2002

4. C1q regulatory region polymorphism down-regulating murine c1q protein levels with linkage to lupus nephritis;Miura-Shimura;J. Immunol.,2002

5. Complement component C1q regulates macrophage expression of Mer tyrosine kinase to promote clearance of apoptotic cells;Galvan;J. Immunol.,2012

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