Heat‐killed BCG induces biphasic cyclooxygenase 2 + splenic macrophage formation—role of IL‐10 and bone marrow precursors
Author:
Affiliation:
1. Department of Biomedical Sciences, Florida Atlantic University, Boca Raton; and
2. Department of Physiology, Brody School of Medicine at East Carolina University, Greenville, North Carolina; and
3. Caswel Beach, North Carolina
Funder
National Institutes of Health
U.S. Department of Defense
Publisher
Wiley
Subject
Cell Biology,Immunology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1189/jlb.1205737
Reference42 articles.
1. Prostaglandin-E2 is a potent inhibitor of human interleukin 12 production.
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3. Rat macrophage treatment with lipopolysaccharide leads to a reduction in respiratory burst product secretion and a decrease in NADPH oxidase affinity
4. Regulation of TNFα and interleukin-10 production by prostaglandins I2 and E2: studies with prostaglandin receptor-deficient mice and prostaglandin E-receptor subtype-selective synthetic agonists
5. Splenic PGE2-releasing macrophages regulate Th1 and Th2 immune responses in mice treated with heat-killed BCG
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