Endotoxin-induced uveitis is primarily dependent on radiation-resistant cells and on MyD88 but not TRIF

Author:

Kezic J.,Taylor S.,Gupta S.,Planck S. R.,Rosenzweig H. L.,Rosenbaum J. T.

Funder

National Institutes of Health

Oregon Clinical and Translational Research Institute (OCTRI)

National Center for Research Resources (NCRR)

NIH Roadmap for Medical Research

Research to Prevent Blindness

William C. Kuzell Foundation

William and Mary Bauman Foundation

Stan and Madelle Rosenfeld Family Trust

American College of Rheumatology and Research to Prevent Blindness

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Reference36 articles.

1. TLR-4/MD2 has a critical role on endotoxin-induced uveitis (EIU);Brito;Invest. Ophthalmol. Vis. Sci.,2008

2. Nucleotide oligomerization domain-2 (NOD2)-induced uveitis: dependence on IFN-γ;Rosenzweig;Invest. Ophthalmol. Vis. Sci.,2009

3. CARD15 mutations in Blau syndrome;Miceli-Richard;Nat. Genet.,2001

4. Association of TLR7 copy number variation with susceptibility to childhood-onset systemic lupus erythematosus in Mexican population;García-Ortiz;Ann. Rheum. Dis.,2010

5. Inhibition of Toll-like receptor 4 breaks the inflammatory loop in autoimmune destructive arthritis;Abdollahi-Roodsaz;Arthritis Rheum.,2007

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