Neutrophil elastase activity compensates for a genetic lack of matrix metalloproteinase-9 (MMP-9) in leukocyte infiltration in a model of experimental peritonitis

Author:

Kolaczkowska Elzbieta,Grzybek Weronika,van Rooijen Nico,Piccard Helene,Plytycz Barbara,Arnold Bernd,Opdenakker Ghislain

Funder

E. K.

“Geconcerteerde OnderzoeksActies”

Fund for Scientific Research of Flanders (FWO-Vlaanderen)

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Reference54 articles.

1. Role of resident peritoneal macrophages and mast cells in chemokine production and neutrophil migration in acute inflammation: evidence for an inhibitory loop involving endogenous IL-10;Ajuebor;J. Immunol.,1999

2. Gelatinase B/MMP-9 as an inflammatory marker enzyme in mouse zymosan peritonitis: comparison of phase-specific and cell-specific production by mast cells, macrophages and neutrophils;Kolaczkowska;Immunobiology,2008

3. Mast cell involvement in zymosan-induced peritonitis in C57Bl/6 mice;Kolaczkowska;Centr. Eur. J. Immunol.,2008

4. Role of lymphocytes in the course of murine zymosan-induced peritonitis;Kolaczkowska;Inflamm. Res.,2008

5. Gelatinase B/matrix metalloproteinase-9 contributes to cellular infiltration in a murine model of zymosan peritonitis;Kolaczkowska;Immunobiology,2006

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