An arrestin-dependent multi-kinase signaling complex mediates MIP-1β/CCL4 signaling and chemotaxis of primary human macrophages-Reference-Cited by-同舟云学术

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An arrestin-dependent multi-kinase signaling complex mediates MIP-1β/CCL4 signaling and chemotaxis of primary human macrophages

Published:2009-07-20 Issue:4 Volume:86 Page:833-845
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:Journal of Leukocyte Biology

Author:

Cheung Ricky,Malik Mobeen,Ravyn Vipa,Tomkowicz Brian,Ptasznik Andrzej,Collman Ronald G.

Funder

National Institutes of Health

Canadian Institutes of Health Research

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Reference72 articles.

1. HIV-1 gp120 and chemokine activation of Pyk2 and mitogen-activated protein kinases in primary macrophages mediated by calcium-dependent, pertussis toxin-insensitive chemokine receptor signaling;Del Corno;Blood,2001

2. HIV-1 gp120-induced TNF-{α} production by primary human macrophages is mediated by phosphatidylinositol-3 (PI-3) kinase and mitogen-activated protein (MAP) kinase pathways;Lee;J. Leukoc. Biol.,2005

3. The Src kinase Lyn is required for CCR5 signaling in response to MIP-1β and R5 HIV-1 gp120 in human macrophages;Tomkowicz;Blood,2006

4. Signaling mechanism of HIV-1 gp120 and virion-induced IL-1β release in primary human macrophages;Cheung;J. Immunol.,2008

5. β-Arrestins and heterotrimeric G-proteins: collaborators and competitors in signal transduction;Defea;Br. J. Pharmacol.,2008

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