The Role of Barrier Dysfunction and Change of Claudin Expression in Inflammatory Bowel Disease
Author:
Affiliation:
1. Department of Internal Medicine and Institute of Gastroenterology, Yonsei University College of Medicine, Seoul, Korea
Publisher
The Editorial Office of Gut and Liver
Subject
Gastroenterology,Hepatology
Link
http://pdf.medrang.co.kr/ekjg/ekjg009-06-04.pdf
Reference11 articles.
1. Intestinal permeability, leaky gut, and intestinal disorders;Hollander, D;Curr Gastroenterol Rep,1999
2. Importance of disrupted intestinal barrier in inflammatory bowel diseases;Salim, SY;Inflamm Bowel Dis,2011
3. Molecular physiology and patho-physiology of tight junctions. IV. Regulation of tight junctions by extracellular stimuli: nutrients, cytokines, and immune cells;Nusrat, A;Am J Physiol Gastrointest Liver Physiol,2000
4. Structure and function of claudins;Krause, G;Biochim Biophys Acta,2008
5. Changes in the expression and distribution of claudins, increased epithelial apoptosis, and a mannan-binding lectin-associated immune response lead to barrier dysfunction in dextran sodium sulfate-induced rat colitis;Yuan, B;Gut Liver,2015
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