Metabolic Consequences of Adenosine Deaminase Deficiency in Mice Are Associated with Defects in Alveogenesis, Pulmonary Inflammation, and Airway Obstruction
Author:
Affiliation:
1. Department of Biochemistry and Molecular Biology, University of Texas Health Science Center at Houston Medical School, Houston, Texas 77030
2. Department of Biochemistry and Molecular Biology, Mayo Clinic, Scottsdale, Arizona 85259
Abstract
Publisher
Rockefeller University Press
Subject
Immunology,Immunology and Allergy
Link
http://rupress.org/jem/article-pdf/192/2/159/1128747/991490.pdf
Reference56 articles.
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2. Adenosine receptor subtypescharacterization and therapeutic regulation;Olah;Annu. Rev. Pharmacol. Toxicol.,1995
3. In vivo inactivation of erythrocyte S-adenosylhomocysteine hydrolase by 2′-deoxyadenosine in adenosine deaminase–deficient patients;Hershfield;J. Clin. Invest.,1979
4. Interference with thymocyte differentiation by an inhibitor of S-adenosylhomocysteine hydrolase;Benveniste;J. Immunol.,1995
5. p53 expression is required for thymocyte apoptosis induced by adenosine deaminase deficiency;Benveniste;Proc. Natl. Acad. Sci. USA.,1995
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