Selective Expression of a Stable Cell Surface Molecule on Type 2 but Not Type 1 Helper T Cells

Author:

Xu Damo1,Chan Woon Ling1,Leung Bernard P.1,Huang Fang-ping1,Wheeler Rachel1,Piedrafita David1,Robinson John H.1,Liew Foo Y.1

Affiliation:

1. From the Department of Immunology, University of Glasgow, Glasgow G11 6NT, United Kingdom; the Department of Virology, St. Bartholomew and Royal London School of Medicine, QMW College, University of London, London EC1A 7BE, United Kingdom; and the Department of Immunology, University of Newcastle, Newcastle Upon Tyne NE2 4HH, United Kingdom

Abstract

T helper cell type 1 (Th1) and 2 (Th2) are central to immune regulation. However, no stable cell surface marker capable of distinguishing and separating these two subsets of CD4+ cells has yet been found. Using differential display PCR, we have identified a gene encoding a cell membrane bound molecule, originally designated ST2L, T1, DER4, or Fit, expressed constitutively and stably on the surface of murine Th2s, but not Th1s even after stimulation with a range of immunological stimuli. Antibody against a peptide derived from ST2L strongly and stably labeled the surface of cloned Th2s but not Th1s, and Th2s but not Th1s derived from naive T cells of ovalbumin T cell receptor–α/β transgenic mice. Three-color single cell flow cytometric analysis shows that cell surface ST2L coexpressed with intracellular interleukin (IL)-4, but not with interferon (IFN)-γ. The antibody selectively lysed Th2s in vitro in a complement-dependent manner. In vivo, it enhanced Th1 responses by increasing IFN-γ production and decreasing IL-4 and IL-5 synthesis. It induced resistance to Leishmania major infection in BALB/c mice and exacerbated collagen-induced arthritis in DBA/1 mice. Thus, ST2L is a stable marker distinguishing Th2s from Th1s and is also associated with Th2 functions. Hence, it may be a target for therapeutic intervention.

Publisher

Rockefeller University Press

Subject

Immunology,Immunology and Allergy

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