THE FUNCTIONAL STATE OF THE RAT HEART DURING ISCHEMIA-REPERFUSION, THE SENSITIVITY OF CALCIUM-INDUCED MITOCHONDRIAL PERMEABILITY TRANSITION PORE OPENING AND THE UNCOUPLING PROTEIN 3 EXPRESSION FOLLOWING LONG EXERCISE TRAINING

Author:

Chorna S.V., ,Talanov S.O.,Strutynska N.A.,Vavilova G.L.,Kotsuruba A.V.,Gaidai N.M.,Sagach V.F., , , , , ,

Publisher

National Academy of Sciences of Ukraine (Co. LTD Ukrinformnauka)

Subject

Physiology

Reference28 articles.

1. Goshovskaya Yu.V., Lisovyi OO, Shymanska TV, Sagach VF Changes in UCP2 and UCP3 Gene Expression, Myocardial Functional Status and Oxygen Cost in Aging and Ischemia-Reperfusion . Fiziol zh.  2009 55, No. 3 P. 26-36.

2. Sagach VF, Vavilova GL, Rudik OV etc. Inhibition of mitochondrial pore as one of the mechanisms of cardioprotective effect of coenzyme Q . Fiziol zh.  2007 53, N 4 P. 35-42.

3. Sagach VF, Rudik OV, Vavilova GL etc. Melatonin Restores Ischemic Tolerance and Reduces the Sensitivity of Mitochondrial Pore Opening in the Heart of Old Rats . Fiziol zh.  2006 52, N 3 P. 3-14.

4. Sagach. VF, Vavilova GL, Rudik OV, Strutinskaya NA. Release of unidentified substances of mitochondrial origin indicators of opening of mitochondrial pore of rat heart . Fiziol zh.  2003 49, N 5 P. 3-12.

5. Sagach. VF, Shimanskaya TV, Nadtochy SM The factor released during the refraction of ischemic heart may be a marker of mitochondrial pore opening . Fiziol zh.  N 4 P. 6-12.

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