Hexamethylbisacetamide and Disruption of Human Immunodeficiency Virus Type 1 Latency in CD4+T Cells
Author:
Publisher
Oxford University Press (OUP)
Subject
Infectious Diseases,Immunology and Allergy
Link
http://academic.oup.com/jid/article-pdf/197/8/1162/18052415/197-8-1162.pdf
Reference50 articles.
1. Long-term follow-up studies confirm the stability of the latent reservoir for HIV-1 in resting CD4+ T cells
2. Presence of an inducible HIV-1 latent reservoir during highly active antiretroviral therapy
3. Effect of interleukin-2 on the pool of latently infected, resting CD4+ T cells in HIV-1-infected patients receiving highly active anti-retroviral therapy
4. Intensification and Stimulation Therapy for Human Immunodeficiency Virus Type 1 Reservoirs in Infected Persons Receiving Virally Suppressive Highly Active Antiretroviral Therapy
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1. Hexamethylene bisacetamide impairs NK cell-mediated clearance of acute T lymphoblastic leukemia cells and HIV-1-infected T cells that exit viral latency;Scientific Reports;2019-03-13
2. Current Strategies for Elimination of HIV-1 Latent Reservoirs Using Chemical Compounds Targeting Host and Viral Factors;AIDS Research and Human Retroviruses;2019-01
3. Contrasting effect of the latency-reversing agents bryostatin-1 and JQ1 on astrocyte-mediated neuroinflammation and brain neutrophil invasion;Journal of Neuroinflammation;2017-12
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