Altered Drug Sensitivity, Fitness, and Evolution of Human Immunodeficiency Virus Type 1 withpolGene Mutations Conferring Multi‐Dideoxynucleoside Resistance
Author:
Publisher
Oxford University Press (OUP)
Subject
Infectious Diseases,Immunology and Allergy
Link
http://academic.oup.com/jid/article-pdf/177/5/1207/2405458/177-5-1207.pdf
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1. The HIV-1 Reverse Transcriptase A62V Mutation Influences Replication Fidelity and Viral Fitness in the Context of Multi-Drug-Resistant Mutations;Viruses;2018-07-19
2. HIV-1 with HBV-associated Q151M substitution in RT becomes highly susceptible to entecavir: structural insights into HBV-RT inhibition by entecavir;Scientific Reports;2018-01-26
3. Structural Insights into HIV Reverse Transcriptase Mutations Q151M and Q151M Complex That Confer Multinucleoside Drug Resistance;Antimicrobial Agents and Chemotherapy;2017-06
4. A Cinnamon-Derived Procyanidin Compound Displays Anti-HIV-1 Activity by Blocking Heparan Sulfate- and Co-Receptor- Binding Sites on gp120 and Reverses T Cell Exhaustion via Impeding Tim-3 and PD-1 Upregulation;PLOS ONE;2016-10-27
5. Population-based human immunodeficiency virus 1 drug resistance profiles among individuals who experienced virological failure to first-line antiretroviral therapy in Henan, China during 2010–2011;AIDS Research and Therapy;2015-06-27
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