KCNQ2 Potassium Channel Epileptic Encephalopathy Syndrome: Divorce of an Electro-Mechanical Couple?
Author:
Publisher
SAGE Publications
Subject
Neurology (clinical)
Link
http://journals.sagepub.com/doi/pdf/10.5698/1535-7511-12.4.150
Reference10 articles.
1. Driving With No Brakes: Molecular Pathophysiology of Kv7 Potassium Channels
2. The mechanism of action of retigabine (ezogabine), a first-in-class K+ channel opener for the treatment of epilepsy
3. Ohtahara syndrome: With special reference to its developmental aspects for differentiating from early myoclonic encephalopathy
4. Voltage Sensor of Kv1.2: Structural Basis of Electromechanical Coupling
5. Determination of the subunit stoichiometry of a voltage-activated potassium channel
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1. Plural molecular and cellular mechanisms of pore domainKCNQ2encephalopathy;2024-01-05
2. Identifying the mechanism of action of the Kv7 channel opener, retigabine in the treatment of epilepsy;Neurological Sciences;2023-07-13
3. The influence of genetics on epilepsy syndromes in infancy and childhood;Acta Epileptologica;2022-10-26
4. Dyshomeostatic modulation of Ca2+-activated K+ channels in a human neuronal model of KCNQ2 encephalopathy;eLife;2021-02-05
5. Clinical characteristics of KCNQ2 encephalopathy;Brain and Development;2021-02
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