CTLA4Ig: a novel inhibitor of costimulation
Author:
Affiliation:
1. Division of Rheumatology, University of California San Francisco, San Francisco, USA
2. Division of Rheumatology, University of California San Francisco, San Francisco, USA,
Abstract
Publisher
SAGE Publications
Subject
Rheumatology
Link
http://journals.sagepub.com/doi/pdf/10.1191/0961203303lu1029oa
Reference21 articles.
1. Lymphoproliferative Disorders with Early Lethality in Mice Deficient in Ctla-4
2. The Role of T-Cell Costimulatory Activation Pathways in Transplant Rejection
3. Costimulatory B7 Molecules in the Pathogenesis of Infectious and Autoimmune Diseases
4. CTLA-4 is a second receptor for the B cell activation antigen B7.
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